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Atherosclerosis is exacerbated by chitinase-3-like-1 in an amyloid precursor protein transgenic mice
- At: 2017 FIP Congress in Seoul (South Korea)
- Type: Poster
- By: SON, Dong Ju (Chungbuk National University, College of Pharmacy, Cheongju, Korea, Republic Of)
- Co-author(s): Dong Ju Son:
Young Sik Seo: Department of Molecular Biology and Institute of Nanosensor Biotechnology, Dankook University, Yongin, Korea, Republic Of
Heonyong Park: Department of Molecular Biology and Institute of Nanosensor Biotechnology, Dankook University, Yongin, Korea, Republic Of
Dae Youn Hwang: College of Natural Resources & Life Science, Pusan National University, Miryang, Korea, Republic Of
Sang Bae Han: College of Pharmacy, Chungbuk National University, Cheongju, Korea, Republic Of
Sanghyeon Kim: Stanley Brain Research Laboratory, Stanley Medical Research Institute, Rockville, United States
Jin Tae Hong: College of Pharmacy, Chungbuk National University, Cheongju, Korea, Republic Of
BackgroundAlthough the critical role of amyloid precursor protein (APP) on vascular diseases associated with Alzheimer’s disease (AD) has been demonstrated, the underlying molecular mechanisms and physiological consequences are unclear.
PurposeWe aimed to evaluate vascular inflammation and atherosclerosis in Swedish mutant of human APP transgenic.. The access to the whole abstract and the presentation file is available to FIP members and to congress participants of that specific congress.
Last update 4 October 2019