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Trem2 and cd163 expression in lps or il-4 stimulated bv-2 microglial cells treated with hx630 and vitamin d3

  • At: 2017 FIP Congress in Seoul (South Korea)
  • Type: Poster
  • By: DULLA, Yevgeny (Graduate School of Pharmaceutical Sciences, Kumamoto University, Department of Chemico-pharmacological Sciences, Kumamoto City, Japan)
  • Co-author(s): Yevgeny Aster Tubola Dulla: Department of Chemico-pharmacological Sciences, Graduate School of Pharmaceutical Sciences, Kumamoto University, Kumamoto City, Japan;HIGO Program, Program for Leading Graduate Schools, Kumamoto University, Kumamoto City, Japan
    Yuki Kurauchi: Department of Chemico-pharmacological Sciences, Graduate School of Pharmaceutical Sciences, Kumamoto University, Kumamoto City, Japan
    Takahiro Seki: Department of Chemico-pharmacological Sciences, Graduate School of Pharmaceutical Sciences, Kumamoto University, Kumamoto City, Japan
    Akinori Hisatsune: Department of Chemico-pharmacological Sciences, Graduate School of Pharmaceutical Sciences, Kumamoto University, Kumamoto City, Japan;HIGO Program, Program for Leading Graduate Schools, Kumamoto University, Kumamoto City, Japan
    Hiroshi Katsuki: Department of Chemico-pharmacological Sciences, Graduate School of Pharmaceutical Sciences, Kumamoto University, Kumamoto City, Japan
  • Abstract:

    Background

    Microglia are said to be involved in the progression of chronic neurodegenerative inflammatory diseases. Recently, phagocytosis of living neurons has been pointed out to be an event that accelerates the progression of these diseases. TREM2 and CD163 are molecules that are related to the phagocytic activity of microglia.  The retinoid X

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Last update 4 October 2019

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