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Fgf21 maintains glucose homeostasis by mediating the crosstalk between liver and brain during prolonged fasting

  • At: 2014 FIP Congress in PSWC, Melbourne (Australia)
  • Type: Presentation
  • By: LIANG, Qingning (The University of Hong Kong, Medicine, Hong kong, China - Hong-Kong)
  • Co-author(s): Zhong, Ling (The University of Hong Kong, Hong kong, China - Hong-Kong)
    Zhang, Jialiang (The University of Hong Kong, Hong kong, China - Hong-Kong)
    Wang, Yu (The University of Hong Kong, Hong kong, China - Hong-Kong)
    Tse, Hung-Fat (The University of Hong Kong, Hong kong, China - Hong-Kong)
    Wu, Donghai (Guangzhou Institute of Biomedicine and Health, Chinese Academy of Sciences, Guangzhou, China - Hong-Kong)
    Bornstein, Stefan (Department of Medicine, University of Dresden, Dresden, Germany)
    Lam, Karen (The University of Hong Kong, Hong kong, China - Hong-Kong)
    Xu, Aimin (The University of Hong Kong, Hong kong, China - Hong-Kong)
  • Abstract:

    I. Background Hepatic gluconeogenesis is a main source of blood glucose during prolonged fasting, and is orchestrated by endocrine and neural pathways. Fibroblast growth factor 21 (FGF21), a hepatocyte-secreted endocrine factor induced by peroxisome proliferator-pctivated Receptor (PPAR) α during prolonged fasting, has recently been shown to be an

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Last update 4 October 2019

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