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Carbon monoxide bound red blood cells protect the expression of hepatic cytochrome p450 after resuscitation from hemorrhagic shock via inactivation of kupffer cells

  • At: 2014 FIP Congress in PSWC, Melbourne (Australia)
  • Type: Poster
  • By: MARUYAMA, Toru (School of Pharmacy, Kumamoto University, Biopharmaceutics, Kumamoto, Japan)
  • Co-author(s): Ogaki, Shigeru (School of Pharmacy, Kumamoto University, Kumamoto, Japan)
    Taguchi, Kazuaki (Faculty of Pharmaceutical Sciences, Sojo University, Kumamoto, Japan)
    Maeda, Hitoshi (School of Pharmacy, Kumamoto University, Kumamoto, Japan)
    Ishima, Yu (School of Pharmacy, Kumamoto University, Kumamoto, Japan)
    Watanabe, Hiroshi (School of Pharmacy, Kumamoto University, Kumamoto, Japan)
    Otagiri, Masaki (Faculty of Pharmaceutical Sciences, Sojo University, Kumamoto, Japan)
  • Abstract:

    [Purpose] The ischemia/reperfusion (I/R) injury is occurred in several clinical conditions, such as resuscitation from massive hemorrhage. Recent study revealed that cytochrome P450 (CYP) was degraded during I/R. Previously, we demonstrated that CO bound red blood cells (RBC), CO-RBC, protected hepatic CYP activity in hemorrhagic shock and..

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Last update 4 October 2019

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