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Olanzapine suppresses insulin secretion from pancreatic β-cells via blockade of multiple receptors

  • At: 2018 FIP Congress in Glasgow (Scotland)
  • Type: Poster
  • By: NAGATA, Mao (Graduate School of Biomedical & Health Sciences, Hiroshima University, Department of Pharmaceutical Services)
  • Co-author(s): Mao Nagata: Department of Pharmaceutical Services, Graduate School of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan
    Tomoe Nakai: School of Pharmaceutical Sciences, Hiroshima University, Hiroshima, Japan
    Yumika Miura: School of Pharmaceutical Sciences, Hiroshima University, Hiroshima, Japan
    Takashi Tomita: Department of Pharmaceutical Services, Hiroshima University Hospital, Hiroshima, Japan
    Takanori Taogoshi: Department of Pharmaceutical Services, Graduate School of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan;School of Pharmaceutical Sciences, Hiroshima University, Hiroshima, Japan;Department of Pharmaceutical Services, Hiroshima University Hospital, Hiroshima, Japan
    Tomoharu Yokooji: Department of Pharmaceutical Services, Graduate School of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan;School of Pharmaceutical Sciences, Hiroshima University, Hiroshima, Japan
    Yumi Sugimoto: Department of Pharmacology, Himeji Dokkyo University, Hyogo, Japan
    Hiroaki Matsuo: Department of Pharmaceutical Services, Graduate School of Biomedical & Health Sciences, Hiroshima University, Hiroshima, Japan;School of Pharmaceutical Sciences, Hiroshima University, Hiroshima, Japan;Department of Pharmaceutical Services, Hiroshima University Hospital, Hiroshima, Japan
  • Abstract:

    Background

    Olanzapine is a potent antipsychotic agent which blocks multiple receptors such as dopamine, serotonin and histamine receptors. The use of olanzapine is sometime limited due to the induction of hyperglycemia as a side effect.

    Methods

    Expressions of dopamine (D2, D3 and 4), serotonin (5-HT2A, 5-HT2B, 5-HT2C and 5-HT6) and histamine H1

    ..

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Last update 4 October 2019

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